Secondary Effects of Catalytic Diesel Particulate Filters

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Author(s) Wenger, Daniela, Gerecke, Andreas C., Heeb, Norbert V., Zennegg, Markus, Kohler, Martin, Naegeli, Hanspeter, Zenobi, Renato
Publication Type Journal Items, Publication Status: Published
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Title Secondary Effects of Catalytic Diesel Particulate Filters
Subtitle Reduced Aryl Hydrocarbon Receptor-Mediated Activity of the Exhaust
Author(s) Wenger, Daniela
Gerecke, Andreas C.
Heeb, Norbert V.
Zennegg, Markus
Kohler, Martin
Naegeli, Hanspeter
Zenobi, Renato
Journal or Series Title Environmental science & technology
Volume Number 42
Issue Number 8
Start Page 2992
End Page 2998
ISSN 0013-936X
1520-5851
Publisher American Chemical Society
Publication Place Washington, D.C.
Publication Date 2008
Abstract Diesel exhaust contains numerous toxic substances that show different modes of action such as triggering aryl hydrocarbon receptor (AhR)-mediated pathways. We investigated AhR-mediated activity of exhaust generated by a heavy-duty diesel engine operated with or without iron- or copper/iron-catalyzed diesel particulate filters (DPFs). AhR agonists were quantified using the DR-CALUX reporter gene assay (exposure of cells for 24 h). We found 54–60 ng 2,3,7,8-tetrachlorodibenzo-p-dioxin CALUX equivalents (TCDD-CEQs) per m3 of exhaust in unfiltered samples and 6–16 ng TCDD-CEQ m−3 in DPF-treated samples. DPF applications decreased TCDD-CEQ concentrations by almost 90%. Concentrations of known AhR agonists were determined with GC/HRMS and converted to TCDD-CEQ concentrations using compound-specific relative potency values. The analyzed nine polycyclic aromatic hydrocarbons (PAHs) and the 17 2,3,7,8-chlorinated dibenzodioxins/furans (2,3,7,8-PCDD/Fs) contributed only marginally (0.6–1.6%) to the total agonist concentration. However, both DPFs also decreased concentrations of individual PAHs by 70–80%. Variation of the assay exposure time (8, 24, 48, 72, and 96 h) revealed that AhR-mediated activity decreased over time and reached a plateau after 72 h, which was most likely due to biotransformation of AhR agonists by the exposed H4IIE cells. At the plateau, we measured 1–2 ng TCDD-CEQ m−3 in both an unfiltered and a filtered exhaust sample. Our findings show that DPFs are a promising technology to detoxify diesel exhaust regarding compounds with AhR-mediated activity.
DOI 10.1021/es071827x
Additional Notes Received July 24, 2007, Revised manuscript received November 29, 2007, Accepted December 20, 2007, Published on Web 03/05/2008
Document Type Article
Publication Status Published
Language English
Assigned Organisational Unit(s) 03430
Organisational Unit(s)
NEBIS System Number 000033453
Source Database ID PP-40953
FORM-1275307938
WOS-000254890400052
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@article{Wngr2008,
  author = "Wenger, Daniela and Gerecke, Andreas C. and Heeb, Norbert V. and Zennegg, Markus and Kohler, Martin and Naegeli, Hanspeter and Zenobi, Renato",
  title = "{S}econdary {E}ffects of {C}atalytic {D}iesel {P}articulate {F}ilters: {R}educed {A}ryl {H}ydrocarbon {R}eceptor-{M}ediated {A}ctivity of the {E}xhaust",
  journal = "Environmental science \& technology",
  year = 2008,
  volume = "42",
  number = "8",
  pages = "2992--2998",
}


E-Citations record created: Thu, 01 Apr 2010, 22:43:40 CET